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Some principles of Somatic Ecology
Dr Erik O. H. Enby, MD, Göteborg 1986.
Translated and edited by Monica Bryant,
The interplay between on one hand all unknown particles and structures,
on the other the tissue cells of the soma in the blood plasma and
the rest of the body fluids, is summarized under the title ”Somatic
Ecology”. If this interplay leads to a state of health or
a disease-state is discussed. In the former case the interplay is
characterized as symbiosis, in the latter as dysbiosis. The importance
of dysbiotic food is brought up for discussion. The degree of severity
of the state of dysbiosis is explained as well as an allopathic
view of the blood.
The term ”somatic ecology” is based on the blood microscopy
research work carried out by Professor Günther Enderlein (1872-1968)
and is supported by more recent microscopic observations of untreated
blood described here.
Professor Enderlein asserted that there are always microorganisms
in blood plasma as well as in the various blood cells and that these
microorganisms can be divided into several different types, which
he was able to identify(1). He also believed that
these microorganisms were always to be found as an integral part of
the life process. They are not only able to divide, but can evolve
through particular stages of development, each with modified properties.
Thus, Enderlein was the foremost advocate of the pleomorphistic theory
According to Enderlein, the majority of these microorganisms spend
their earliest stages of development in a mutually beneficial relationship
with the tissue cells in the soma - this is the so-called stage of
symbiosis(2). In the later developmental stages they
can attack the cells of the different tissues of the soma and this
is known as the stage of dysbiosis. Enderlein asserted that this dysbiosis
can cause the development of somatic disturbances which, in turn,
lead to the individual’s experience that he is gradually moving
from a ”state of health” to a ”state of illness”.
According to Professor Sylvia Silver there is a widespread normal
flora of microorganisms with anaerobic qualities throughout the body(3).
Under certain circumstances they can develop into pathogenic forms
and cause infection. Silver also maintains that a normal reduction-oxidation
potential in a range between 120-150 mV, which is mainly related to
the oxygen contents in the tissues, is probably one of the body’s
major defences against infections caused by pathogenic development
of the normal anaerobic flora, as well as by other facultative anaerobes
invading the tissues.
Different techniques which increase the oxygen intake, such as deep
breathing or inhalation of oxygen according to the method of Professor
Ardenne(4) or by using negatively ionized oxygen
and ozone therapy, have for a long time proved to be very effective
in the treatment of a large number of chronic diseases. Organically-bound
germanium also has the ability to raise the oxygen contents of the
tissues and has been shown to be very effective in the treatment of
The following microscopic investigations of untreated blood described
below were carried out based on the knowledge of:
a) The literature concerning microbial flora in the blood.
b) The therapeutic successes using techniques which raise the reduction-oxidation
potential of the tissues.
The blood of two groups of experimental subjects were examined:
a) 60 healthy subjects ranging in age from 20 to 30 years.
b) 500 patients with chronic illnesses taken from my practice, who
had already been diagnosed at the hospital. The types of patients
involved were primarily those with severe allergies, neurological
diseases, skin and muscle diseases, and different types of malignant
Leitz’ laboratory microscope Dialux 20 equipped with a 100W
halogen lamp. Modified UK condenser for darkfield, lightfield and
interference contrast, Plan-Fluotar-objective, and a binocular photo
tube FSA. All documentation was made with Leitz’ completely
automatic microscope camera, Vario-Orthomat.
specimens for microscopy
Blood from the fingertips of the experimental subjects. The drop of
blood was smeared out by capillary action to form a thin layer between
the cover slide and the object slide. In order to prevent drying,
the edges of the cover slide were covered with immersion oil. Microscopy
work was carried out immediately or within four hours of the specimens
In two earlier studies(6, 7) it
is shown that all the blood examined contained microbe-like formations
and that they are found both in the cells and in the plasma. In
several cases it has been possible to observe them for several weeks
in a thin plasma film between two glass- slides and notice that
they exist in a great number of different forms and sizes. In most
cases the smallest forms were found in the blood of healthy individuals;
the forms often proved to be larger and in a greater quantity in
blood from persons with serious states of diseases.
In observing the microbe-like formations in blood from corpses,
all the microorganisms which can be seen in a living individual’s
blood still show the same amount of activity. They often had increased
further in size, and often there was a sudden emergence of very
small and rapidly mobile forms (not equivalent to the Brownian movement).
Initially, it was difficult to prove that these particles really
were microorganisms as Enderlein maintained. However, it was possible
over a period of a few days to follow these microbe-like formations
and photograph them microscopically.
This showed that they exhibit remarkable properties, such as suddenly
emerging from apparently nowhere and then developing into a variety
of new and varied forms. This might be a proof that these observed
particles really are living units…microorganisms. Enderlein
also stated that they can be cultivated and develop from very small
forms 0,01µm (virus forms) into bacterial forms and further
into fungal forms which are found in the tissues during their degeneration
following the death of the individual.
It is therefore postulated here that these microbe-like formations
in blood really are microorganisms. According to Enderlein’s
theory, the smallest microbial forms behave in symbiosis with the
tissue cells. The higher developed forms, however, are able to attack
and destroy the tissue cells. This process results in an increasing
dysbiosis. The differing states of symbiosis and dysbiosis within
the soma will be called ”the interplay in the soma”.
The interplay between microorganisms and tissue cells in the soma
always takes place in a fluid medium consisting of blood plasma
and interstitial fluid. However, in our fixed tissues such as muscles,
nerves and organs etc, all cells are interconnected. These tissue
cells are surrounded by the interstitial fluid in exactly the same
way as the blood plasma surrounds blood cells.
From this fact it becomes evident that the structure of the blood
is, in principle, similar to that of the fixed tissues. The essential
difference is that blood is a form of fluid tissue. It could therefore
be argued that, if Enderlein’s discovery of microorganisms
in the blood is correct, then similar microbial conditions can also
be found in our fixed tissues.
This understanding of the relationship between the blood and the
tissues reveals that there is a continuity of microbial activity
throughout the soma as a whole. This continuity is, however, not
to be confused with uniformity. The microbial interplay within the
soma is far from homogeneous. In the first place, there are gradients
in the density of their occurrence throughout the soma. Secondly,
there is a difference between the ecological properties of the microorganisms,
either symbiotic or dysbiotic.
oxygenation and circulation
Within the monomorphistic microbiology of today, it is assumed that
microorganisms multiply solely by division, that bacteria gradually
increase in size and then divide to create two new bacteria. The
pleomorphists, however, maintain that this means of propagation
in a bacterial culture only takes place when the culture is constantly
and gradually nourished. When nourishment is lacking, microorganisms
in a medium do not cease to exist, but rather evolve towards other
forms of increasing size with variable properties and characteristics.
It is likely that a decrease in circulation of the body fluids in
part of or throughout the whole soma over a period of time would
result in a decreased supply of’ nourishment and of oxygen
to those parts of the soma. This same principle of depleted nourishment,
in its extreme, occurs in the stagnation of the body fluids of a
A result of this deficiency would be an increase in the microbes’
aggressive behaviour towards their environment, which in this case
are the tissue cells. According to the pleomorphistic way of thinking,
the microorganisms develop into other forms, able to consume and
destroy the tissue cells, first causing diseases and then later
the putrefaction of the soma(8).
Good oxygenation is important because it normalizes the pH in the
tissues(9). This contributes to keeping the microbial
flora at a symbiotic level. A similar effect on the pH in the blood
also occurs through a predominantly alkaline-forming diet. Through
a good circulation, oxygenation and nourishment supply, it is possible
to counteract dysbiotic development.
It therefore becomes evident why increased breathing (increased
oxygen supply) and increased pulse rate (increased circulation),
together with appropriate nutrition are such important factors in
bringing an individual’s soma from a state of dysbiosis (illness)
into a state of symbiosis (health).
In this connection it is interesting to note the result of Ardenne’s
research on the influence of infections on the oxygen partial pressure
of the arterial blood. This pressure is normally about 97 mmHg,
but in elderly people (70 years) it decreases to an average value
of 70 mmHg. This pressure can be lowered to an average of 25 mmHg
by infection and also by other stress factors, such as radiation
treatment, surgical trauma and long durations without movement.
In this case the minimal partial pressure of oxygen becomes increasingly
lower, the lower the initial oxygen pressure of the patient.
The oxidation-reduction potential in the tissues of elderly people
can become very low because of many reasons and this explains, partly,
the increasing tendency to develop increased severity of disease
in the later years of life.
It is likely that the principles of somatic ecology, described so
far, are equally relevant to all the life processes throughout nature.
The pleomorphistic developmental stages, towards more pathogenic forms,
occur whenever the environmental conditions are conducive in any tissue,
whether plants or animals(10).
It is important to realize that the soma of an individual is in a
dynamic interaction with the external environment. There is an intimate
exchange of intake and output of inanimate and animate substances.
For example, the quality of the life process in the form of food consumed
by an individual will have a great influence on its somatic ecology
as it becomes an integral part of the person.
Daily environmental factors therefore play a critical role in the
maintenance of a symbiotic state or the development of a dysbiotic
state within the soma. The consumption of food in dysbiosis will influence
the consumer’s quality of life in a negative way. For example,
to eat much of the food which today is depleted of quality due to
its growth under artificial conditions is dysbiosis-forming.
During my microscopy work I have been able to find evidence to support
everything which was presented by Enderlein in his book Bakterien-Cyclogenie(1).
I also verified the occurrence of microorganisms in the blood of chronically
ill people which have not been described previously in the literature
of allopathic or alternative medicine(6, 7).
If microbial masses, present in the blood, occur in the solid tissues,
they may be responsible for different manifestations of disease through
their polymorphic alterations of the tissue cells. Such histological
changes of the fixed tissue are particularly evident in malignant
These microorganisms in the body fluids are, most likely a necessary
component in the life process which probably cannot function or ”flow
on” without them. They have adapted to the different cell types
in the soma, so that they can exist in either a state of symbiosis
or dysbiosis, depending upon the environmental conditions of the soma.
The severity of dysbiotic disturbance depends on the type of cells
involved, as well as the degree of pathogenic behaviour displayed
by these microorganisms towards these tissue cells. For example, if
the red blood corpuscles were attacked primarily by aggressive microorganisms,
the infection would lower the partial oxygen pressure of the blood
and, in addition to this, anaemia would gradually develop and further
lower the partial oxygen pressure. A lowered oxidation-reduction-potential
causes a multiplication of the anaerobic bacteria throughout the whole
organism. The result of this development would, in course of time,
lead to a more general attack on many other tissue cells of the soma.
According to this reasoning, a condition such as leukaemia might not
be considered a primary disturbance of the white blood corpuscles.
Their increase in the blood, which is also sometimes observed in the
course of anaemia, can be explained as the soma’s defence against
the aggressive attack of the microorganisms. The intracellular attack
which can frequently be observed in the red blood corpuscles in this
type of somatic disturbance is probably only the easily visible part
of the entire intracellular attack taking place in the soma, during
the development of these serious symptoms.
This could explain why patients with degenerative diseases can sometimes
lose up to 15-20 kilograms in weight, along with the dramatic deterioration
in general condition which frequently occurs and cannot be explained
solely by a decrease in haemoglobin count. Perhaps a more general
attack upon the soma’s tissue cells can also explain why leukaemia
patients die suddenly despite the fact that the haemoglobin count
is, at death, still compatible with continued life. If, instead, these
microorganisms are aimed primarily at cell types which are less important
for overall survival of the individual and not crucial for the maintenance
of a normal circulation and internal milieu in the soma, other disturbances
occur which certainly decrease the quality of life for the individual
but do not pose an immediate threat to it.
Professor Enderlein’s results showed that some of the microorganisms
he observed in the blood could be structurally transformed by the
same microorganisms at earlier developmental stages. By using this
biological phenomenon, it is possible to decrease the aggressive activity
of the microorganisms in the soma and even make them harmless to its
The administration of symbiogenic remedies, which contain dilutions
of the transforming microbial particles, to a soma in a state of dysbiosis,
can bring about the fundamental cure of a disease. This is the core
of what Enderlein showed: that it is possible to regulate a dysbiotic
interplay by introducing to the soma symbiosis-producing microorganisms.
He developed remedies for the treatment of both acute and chronic
degenerative somatic diseases which are used in conjunction with the
regulation of pH and increasing circulation, nutrient and oxygen intake.
Through this kind of treatment, patients with troublesome and fatal
chronic diseases are able to have astonishing improvements. The restructuring
process in the soma ceases and a state of ”health” returns,
and the patients seldom suffer relapses after the treatment is completed.
Allopathic pharmacological forms of treatment mainly camouflage symptoms,
while the symptom-producing, dysbiotic interplay in the soma progresses
in an uninterrupted way. This results in an increase of symptoms and
a further need for pharmacological symptom-suppressing preparations.
Enderlein’s somatic, reharmonizing, biological form of therapy,
”Symbiosis Therapy”, has very high success rate. It would
be rewarding to carry out further investigations into states of diseases
which have not yet responded well. This is probably because Enderlein
in his lifetime did not manage to describe in detail all the various
types of processes in the ubiquitous particle-cell interplay which
occurs in the soma. Undoubtedly, there are a number of other kinds
of microorganisms which have to be identified. A continued study of
microbial particles in the soma unknown so far, and their ability
to alter the living substance, will gradually help us to understand
how a multitude of different chronic, troublesome and serious illnesses
arise. This could enable Enderlein’s method of treatment to
reach its full potential in dealing with the entire panorama of diseases
which are based on forms of dysbiotic interplay.
view of microorganisms
It is remarkable that the microorganisms in the blood are so far unrecognized
within allopathic medicine. This may be because all haematological
research carried out in the past has employed microscopy methods that
make it impossible to see these microorganisms in the plasma and blood
Many attempts have been made in medical microbiology to cultivate
the kinds of micoorganisms which may occur in the blood. In the case
of various chronic illnesses these investigations have not met with
success, and gradually allopathic medicine began to believe that most
chronic illnesses were attributable either to chromosomal changes
or to chemical disturbances in the cells. The reason why such cultivations
have been unsuccessful, despite the fact that blood is full of microorganisms
in all conditions of chronic illness, is probably the use of inappropriate
An important reason why virtually no researcher has thought of examining
the presence of microorganisms in the blood from patients with different
chronic diseases may be that within conventional haematological research
it is assumed a priori that blood
is sterile. This idea has continued to dominate the thinking within
orthodox medicine and prevents research into the possible existence
of microbial life in the blood.
(1) Enderlein, Günther
(1981). Bakterien-Cyclogenie. (2.
Ausgabe). Hoya. Semmelweis-Verlag.
(2) Enderlein, Günther
(1955). Akmon. Das Blut als Phaenomen
der Gesundheit in akmosophischer Betrachtung. Ibica-Verlag.
(3) Silver, S.
(1980). Anaerobic Bacteriology for the
Clinical Laboratory. C.V. Mosby Company.
(4) Ardenne M. V., Wiemuth H. H. and Wiesner S.
(1980). Messungen über permanente
bzw. zeitweilige Steigerung der arterio-venoesen p02-Differenz durch
den 02-Mehrschritt-Regenerationsprozess bzw. Reinfusion von UV-bestrahltem
Eigenblut. Deutsche Gesundheitswesen. No 35. p 1620-1629.
(5) Asai, Kazuhiko
(1980). Organisches Germanium. Eine Hoffnung
für viele Kranke. Hoya. Semmelweis-Verlag.
(6) Enby, Erik O. H.
(1984). Mikrobliknande bildningar i blod
vid kroniska sjukdomar. (Microbe-like
formations in the blood of patients with chronic diseases).
Svensk Tidskrift för Biologisk Medicin, Swedish Journal of
Biological Medicine. No 1. p 22-26.
(7) Enby, Erik O. H.
(1983). Redovisning av fynd vid mikroskopering
av levande blod från två patienter med Morbus Hodgkin
och tre patienter med maligna tumörsjukdomar. (Report
of findings from microscopic examination of fresh blood from two
patients with Hodgkin's disease and three patients with malignant
tumours). Göteborg. Edition C&L Förlag.
(8) Atlas, R. M.
(1984). Microbiology: fundamentals and
application. New York. Macmillan.
(9) Szilvay, Gyula de
(1981). Grundlagenforschung über
Krebs und Leukämie. (2. Auflage). Hoya. Semmelweis-Verlag.
(10) Tissot, J.
(1926). Constitution des organismes animaux
et végétaux. Causes des maladies qui les atteignent.
Band 1-3. Laboratoire de physiologie générale du Muséum
d’histoire naturelle. Paris.
© 1986-2004. Dr Erik Enby. All rights reserved. This article
may only be reproduced in its entirety.
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